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51207-85-3

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51207-85-3 Usage

General Description

Benzamide, 4-amino-N,N-diethyl- is a chemical compound with the molecular formula C11H16N2O. It is an amide derivative of benzoic acid, and it is known for its pharmaceutical and industrial applications. Benzamide, 4-amino-N,N-diethyl- is commonly used as a precursor in the synthesis of various pharmaceutical drugs and dyes. Its chemical structure and properties make it suitable for use in the production of medicines, pesticides, and other organic compounds. 4-amino-N,N-diethyl-benzamide is also used as a reactant in organic synthesis and is an important building block for the creation of complex chemical compounds.

Check Digit Verification of cas no

The CAS Registry Mumber 51207-85-3 includes 8 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 5 digits, 5,1,2,0 and 7 respectively; the second part has 2 digits, 8 and 5 respectively.
Calculate Digit Verification of CAS Registry Number 51207-85:
(7*5)+(6*1)+(5*2)+(4*0)+(3*7)+(2*8)+(1*5)=93
93 % 10 = 3
So 51207-85-3 is a valid CAS Registry Number.
InChI:InChI=1/C11H16N2O/c1-3-13(4-2)11(14)9-5-7-10(12)8-6-9/h5-8H,3-4,12H2,1-2H3

51207-85-3SDS

SAFETY DATA SHEETS

According to Globally Harmonized System of Classification and Labelling of Chemicals (GHS) - Sixth revised edition

Version: 1.0

Creation Date: Aug 14, 2017

Revision Date: Aug 14, 2017

1.Identification

1.1 GHS Product identifier

Product name 4-Amino-N,N-diethylbenzamide

1.2 Other means of identification

Product number -
Other names N,N-Diethyl 4-aminobenzamide

1.3 Recommended use of the chemical and restrictions on use

Identified uses For industry use only.
Uses advised against no data available

1.4 Supplier's details

1.5 Emergency phone number

Emergency phone number -
Service hours Monday to Friday, 9am-5pm (Standard time zone: UTC/GMT +8 hours).

More Details:51207-85-3 SDS

51207-85-3Relevant articles and documents

An Activatable Photosensitizer Targeted to γ-Glutamyltranspeptidase

Chiba, Mayumi,Ichikawa, Yuki,Kamiya, Mako,Komatsu, Toru,Ueno, Tasuku,Hanaoka, Kenjiro,Nagano, Tetsuo,Lange, Norbert,Urano, Yasuteru

, p. 10418 - 10422 (2017)

We adopted a spirocyclization-based strategy to design γ-glutamyl hydroxymethyl selenorhodamine green (gGlu-HMSeR) as a photo-inactive compound that would be specifically cleaved by the tumor-associated enzyme γ-glutamyltranspeptidase (GGT) to generate th

Synergistic Effects of ppm Levels of Palladium on Natural Clinochlore for Reduction of Nitroarenes

Gholinejad, Mohammad,Oftadeh, Erfan,Shojafar, Mohammad,Sansano, José M.,Lipshutz, Bruce H.

, p. 4240 - 4248 (2019/09/06)

Augmenting the modified naturally occurring clay clinochlore with ppm amounts of palladium leads to a new and very effective reagent for the reduction of numerous aromatic nitro species. When palladium nanoparticles are supported on pyridyltriazole-modified clinochlore, iron within clinochlore acts synergistically with palladium to catalyze the reduction of a wide variety of nitroarenes at room temperature in aqueous media. Based on E-factor calculations, the catalyst system is found to be in line with green chemistry standards and can be recycled up to five times.

Design, synthesis and biological evaluation of novel 2-phenyl pyrimidine derivatives as potent Bruton's tyrosine kinase (BTK) inhibitors

Li, Xinyu,Shi, Binyu,Teng, Yu,Cheng, Yu,Yang, Huizhu,Li, Jiurong,Wang, Lianjian,He, Siying,You, Qidong,Xiang, Hua

supporting information, p. 294 - 299 (2019/03/02)

BTK is an effective target for the treatment of B-cell malignant tumors and autoimmune diseases. In this work, a series of 2-phenyl pyrimidine derivatives were prepared and their preliminary in vitro activities on B-cell leukemia cells as well as the BTK enzyme were determined. The results showed that compound 11g displayed the best inhibitory activity on BTK with an inhibition rate of 82.76% at 100 nM and excellent anti-proliferation activity on three B-cell leukemia lines (IC50 = 3.66 μM, 6.98 μM, and 5.39 μM against HL60, Raji and Ramos, respectively). Besides, the flow cytometry analysis results indicated that 11g inhibited the proliferation of the Raji cells in a dose- and time-dependent manner, and blocked the Ramos cells at the G0/G1 phase, which is in accordance with the positive control ibrutinib. The mechanism investigation demonstrated that 11g could inhibit the phosphorylation of BTK and its downstream substrate phospholipase γ2 (PLCγ2). All these results showed that 11g was a promising lead compound that merited further optimization as a novel class of BTK inhibitor for the treatment of B-cell lymphoblastic leukemia.

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